Concise summaries of published swine health and production research, organized for quick discovery.
PPV2 was common in lungs from Colombian pigs, but coinfections predominated and direct localization in lesions was uncommon, so detection did not establish causation.
In this experimental model, DNA from inactivated M. hyopneumoniae did not persist at detectable levels, unlike viable organisms.
In mixtures containing modified-live vaccine and wild-type PRRSV, ORF5 Sanger sequencing primarily represented the dominant viral population and could mask a minority strain.
Exhaust-air sampling detected the onset of PRRSV circulation at the same sampling event as oral fluids in three barns, but it was less sensitive for later monitoring.
Experimentally infected pigs moved less, suggesting that automated movement monitoring may complement conventional measures of M. hyopneumoniae infection.
An mRNA-targeting PCR may better reflect M. hyopneumoniae viability than conventional DNA PCR, particularly near the end of elimination programs.
Longitudinal comparison of four sample types for PRRSV RNA detection in pregnant gilts after live-virus inoculation.
A longitudinal comparison of postmortem tongue fluids and oral fluids for PRRSV and influenza A surveillance in growing pigs.
An evaluation of tongue tips from dead piglets as a potential population sample for PRRSV surveillance.
An evaluation of family oral fluids as a litter-level sample for PRRSV surveillance before weaning.
A comparison of several swab types with serum for PRRSV detection in piglets at weaning.